CURRENT STATUS
The first topical gene therapy β Vyjuvek β is now FDA-approved and applied at home for dystrophic epidermolysis bullosa. Topical and oral JAK inhibitors deliver repigmentation in vitiligo and rapid clearance in atopic dermatitis; TYK2 inhibitors and IL-23 biologics clear psoriasis. AI-guided biomarker work is ending trial-and-error prescribing.
KEY BREAKTHROUGHS
Vyjuvek (beremagene geperpavec) β first FDA-approved topical, redosable gene therapy, delivering COL7A1 for dystrophic epidermolysis bullosa
Ruxolitinib cream (Opzelura) β first topical JAK inhibitor to achieve meaningful repigmentation in vitiligo
Deucravacitinib (Sotyktu) β first oral TYK2 inhibitor for plaque psoriasis
IL-13/IL-4 and IL-23 biologics (dupilumab, risankizumab) transforming atopic dermatitis and psoriasis
AI-COMPRESSED PIPELINE
AI TOOLS ACCELERATING CURES
KEY ORGANIZATIONS
KEY CLINICAL TRIALS
Vyjuvek (B-VEC) Label Expansion & At-Home Gene Therapy
ViewKrystal Biotech
A redosable topical gene therapy using an engineered HSV-1 vector to deliver functional COL7A1 to the skin of people with dystrophic epidermolysis bullosa, restoring type VII collagen and healing chronic wounds β applied at home.
Topical & Oral JAK Inhibitors for Vitiligo Repigmentation
ViewIncyte / academic
Trials extending JAK inhibition (topical ruxolitinib and oral agents), often combined with phototherapy, to drive durable repigmentation across more of the body surface in vitiligo.
TIMELINE ESTIMATE
Psoriasis / atopic dermatitis control & vitiligo repigmentation: available now. Broader gene therapy for genodermatoses: 3β7 years. Durable vitiligo cure: 4β8 years.